Plasmid-based gap-repair recombineered transgenes reveal a central role for introns in mutually exclusive alternative splicing in Down Syndrome Cell Adhesion Molecule exon 4

Irmgard Haussmann, Pinar Ustaoglu, Ulrike Brauer, Yash Hemani, Thomas C Dix, Matthias Soller

    Research output: Contribution to journalArticlepeer-review

    13 Citations (Scopus)
    62 Downloads (Pure)

    Fingerprint

    Dive into the research topics of 'Plasmid-based gap-repair recombineered transgenes reveal a central role for introns in mutually exclusive alternative splicing in Down Syndrome Cell Adhesion Molecule exon 4'. Together they form a unique fingerprint.

    Immunology and Microbiology

    Neuroscience

    Biochemistry, Genetics and Molecular Biology